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Moreover, NAC reestablishes mitochondrial dysfunction by attenuating ROS/RNS levels and restoring enzyme complexes involved in oxidative phosphorylation, thereby preserving an adequate rate of mitochondrial respiration

Monounsaturated fatty acids prevent the deleterious effects of palmitate and high glucose on human pancreatic beta-cell turnover and function

Peptide BPC-157 has been touted as speeding up tissue repair by increasing blood vessels in the body based on an animal study, something scientists have warned could potentially encourage the growth of cancerous or pre-cancerous cells

Further, GLP-1RAs play a Frontiers in Endocrinology | www.frontiersin.org August 2021 | Volume 12 | Article 7211351 Edited by: Kazunori Sango, Tokyo Metropolitan Institute of Medical Science, Japan Reviewed by: Mohammad Qaddoumi, Kuwait University, Kuwait Sarah Louise Gray, Furtwangen University, Germany *Correspondence: Yi Zhang [email protected] Yunfeng Liu [email protected] ORCID: Yi Zhang orcid.org/0000-0003-0305-3127 These authors have contributed equally to this work and share rst authorship These authors have contributed equally to this work Specialty section: This article was submitted to Clinical Diabetes, a section of the journal Frontiers in Endocrinology Received: 06 June 2021 Accepted: 05 August 2021 Published: 23 August 2021 Citation: Zhao X, Wang M, Wen Z, Lu Z, Cui L, Fu C, Xue H, Liu Y and Zhang Y (2021) GLP-1 Receptor Agonists: Beyond Their Pancreatic Effects