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Despite these advances, the role of ferroptosis in cardiovascular disease remains less well characterized than in fields such as oncology and neurodegeneration, where it has been more extensively investigated [9,10,11]

In addition, lactylation of Serpina3k at K351 in cardiac fibroblasts during I/R enhances protein stability, protecting cardiomyocytes from apoptosis

Proper technique improves injection preparation across healthcare environments

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Stabilization of mitochondrial lipid components, particularly cardiolipin, by MTP-131 supplementation, has been showed to restore mitochondrial function in vitro , suggesting new potential therapeutic approaches for vitiligo (Chen et al., 2017)