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This decrease in 3-HSD and 17-HSD might have occurred due to testicular cell injury, as there was a remarkable rise in oxidative stress in the testicular tissue, or it could be the result of CYP's interference with the transcriptional regulation of the 3-HSD and 17-HSD genes

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However, the exact link between TMAO and these diseases remains unclear

So before I hand over to the chair and the expert speakers, I just want to welcome everyone to the comms webinar and just let you know while you're listening to the presenters, if you have any questions, there will be time for the Q&A at the end, so please do use the chat function to ask questions as you think of them and we'll make time at the end to have a discussion about these with the presenters