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Even subclinical thiamine depletion can worsen anxiety, impair glucose regulation, and amplify alcohol-related neurological stress

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Oxidation of cysteine residues can result in a wide range of redox-based, post-translational modifications, including S-nitrosylation, S-glutathionylation and sulfinic acid, sulfenic acid and disulfide formation 22,23 , which can generate conformational changes in protein structure and subsequent modulation of protein activity 24

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2dg), and GSK3beta inhibitors (Fig