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This trade-off is consistent with in human evidence that sustained energetic stress can lead to progressive myelin loss even in the absence of overt axonal pathology (Ramos-Cabrer et al., 2025), suggesting that metabolic compensation may preserve short-term function at the expense of long-term myelin integrity

describe how transcriptomic and proteomic analyses, enhanced by machine learning classifiers like random forests and support vector machines, have identified OA-relevant genes such as RIPK3, PDK1, and CDH2

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