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Safety and Tolerability of Glucagon-Like Peptide-1 Receptor Agonists Utilizing Data from the Exenatide Clinical Trial Development Program

GPX2, a direct target of p63, inhibits oxidative stress-induced apoptosis in a p53-dependent manner

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Hence, augmentation of autophagy function in acute seizure and SE could be a compensatory mechanism to restrict neuronal loss and promote rearrangement of neurons and synaptic instability

TGF-1 evokes human airway smooth muscle cell shortening and hyperresponsiveness via Smad3