To dissect the upstream regulatory mechanism of miR-29b-3p under Gln deprivation conditions, we integrated transcriptome data (GSE148016) from both Gln+ and Gln- groups, and performed cross-analysis with the predicted upstream transcription factors of miR-29b-3p, ultimately identifying five candidate transcription factors
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The work described in this publication benefited from the support of the Metabolomics Shared Resource and of the Biobank Shared Resources (BSR) at the Wilmot Cancer Institute, supported in part by a University of Rochester Wilmot Cancer Institute Support Grant (P30CA272302)