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In animal models of heart failure induced by high-frequency pacing, ischaemia, and obesity, GLP-1RAs inhibit left ventricular remodelling and improve left ventricular contractility and diastolic function through anti-inflammatory effects, cardiomyocyte apoptosis inhibition, and glucose metabolism enhancement in cardiomyocytes [38,39,40]

Theyre best suited to people who are maintaining a healthy diet and exercise regime and are close to their ideal weight, but are struggling with stubborn, hard-to-lose pockets of fat in certain areas

Lower doses may still activate GLP-1 receptors, but appetite suppression tends to scale with dose intensity

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