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These findings indicate that D1R-dependent repression of novel-m009C relieves inhibition of Grin1, enhancing NMDAR signaling to promote METH reward 109

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A prospective clinical study has demonstrated that ferroptosis levels showed considerable promise as a novel monitoring indicator and therapeutic target, as they play a critical role in predicting the healing rate of DFUs and assisting clinical treatment decisions [24]

In AD, decreased AQP4 activity impairs A and tau clearance, partly via reduced LRP1-mediated transport

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