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Scatter factor/hepatocyte growth factor stimulation of glioblastoma cell cycle progression through G(1) is c-Myc dependent and independent of p27 suppression, cdk2 activation, or E2F1-dependent transcription

Recently published data in the journal Sherry Shen, MD, assistant attending physician at Memorial Sloan Kettering Cancer Center, and the lead author on the study, spoke to CancerNetwork about the results from the trial and how she is using that data to propel her research forward

Additionally, inhibition of CD38 in the AD mouse model restored NAD + levels, decreased mitochondrial ROS production and plaque depositions in the hippocampus and cortex, and improved cognitive abilities 158

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