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Discussion In this study, we used structure-function analyses to determine the molecular function of the non-acidic C-terminal residue of the SARS-CoV-2 S protein, revealing the importance of this lack of mimicry of coatomer interacting residues
In the context of DR, this leads to the phosphorylation of ERK1/2, increasing downstream expression of anti-apoptotic genes like BCL2 and reducing the activity of pro-apoptotic markers such as Caspase-3
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