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Therefore, our results demonstrate a subset-specific role for GSH in controlling the redox balance underlying the metabolic properties between MZB and FoB

Moreover, THBS1 can mitigate the menstruation-driven inflammatory response by binding to cell-surface receptors (such as a4b1 integrin) of inflammatory cells, thereby restoring the overall immune homeostasis when proceeding to the next stages of the menstrual cycle [18]

Damaged mitochondria leak excess electrons, causing an explosion of ROS that completely overwhelms the cell's antioxidant capacity

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Moreover, theranostic liposomes, which co-deliver drugs and imaging agents, have been developed to enable simultaneous therapy and real-time monitoring [121]