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To determine whether these transcriptomic changes were reflected at the metabolic level, we quantified -hydroxybutyrate, a terminal product of -oxidation, in both cell lysates and culture supernatants

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These studies have identified specific biomarkers of oxidative damage, elucidated the dysregulation of antioxidant defense systems, mapped connections between oxidative stress and inflammatory pathways, and began to explore the influence of CFTR genotype and modulator therapies on the redox landscape of CF

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