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What made AOD 9604 compelling was the identification that a specific region of the hGH molecule, the C-terminal fragment, was responsible for this fat-burning effect, and that this region operated through a pathway separate from the growth hormone receptor

Phase 2 study of neoadjuvant treatment with NOV-002 in combination with doxorubicin and cyclophosphamide followed by docetaxel in patients with HER-2 negative clinical stage II-IIIc breast cancer

The peptide interacts with growth hormone receptors and stimulates fibroblast activity, producing collagen and repairing damaged tissue