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[DOI] [PubMed] [Google Scholar] 92.Trexel J., Yoon G.S., Keswani R.K., McHugh C., Yeomans L., Banerjee R., Sud S., Sun Y., Rosania G.R., Stringer K.A

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No similar fluorescence was observed with the FITC-labelled 2 o antibody control

Uno de los mecanismos clave en la fisiopatologa de la obesidad es la inflamacin crnica de bajo grado, mediada por la produccin de citocinas proinflamatorias y adipocinas que contribuyen al desarrollo de resistencia a la insulina y a una activacin persistente del sistema nervioso simptico, lo que genera un aumento de la presin arterial y favorece la disfuncin endotelial, incrementando el riesgo cardiovascular