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| | 5-Amino-1MQ Monotherapy | None | 50100mg daily | Daily oral or subcutaneous | NNMT inhibition preserves NAD+, activates AMPK and mitochondrial fat oxidation | 812 weeks | Promising preclinical data

This is an oral compound with a completely distinct mechanism from any GLP-1 receptor agonist.

However, whether the potential resistance of NPCs is an event associated with a compensatory mechanism of AD cells at early stages, or if it also occurs at later stages of differentiation remains to be elucidated

Hemolysis, the type of anticoagulant used, and prolonged storage can artificially increase MDA levels