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Recent studies have documented that GLP-1R engagement in the portal circulation is reduced under high-fat dietfeeding conditions (68), suggesting together with our data that multiple mechanisms converge to control GLP-1 action in the hepatic portal circulation, which may contribute to glucose dysregulation in mice upon high-fat feeding

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Immobilization stress: NaBPC157 10 g/kg and 10 ng/kg, intraperitoneally one hour before and immediately after the tying, and at 24 and 48 hours of immobilization

Appetite suppression: GLP-1 injections work by interacting with the brains appetite control centers

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