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The higher dietary fat intake (9weeks duration) of HFD-fed recipients did not result in differences in percent body weight gain, endocrine parameters (insulin, estradiol, and progesterone), and estrous cyclicity, relative to CD-fed recipients, indicating the preferential contribution of the local peritoneal environment rather than the systemic milieu to underlie the differences in lesion parameters

Altogether, we conclude that HSV-1 infection can enhance Keap1-dependent Nrf2 degradation, thereby disrupting cellular redox homeostasis and promoting ferroptosis