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As dementia and more severe symptoms occur during the course of AD, other molecular pathways may also be involved in AD progression, thus requiring the use of additional drugs

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Our data suggest that GCLM KO mice have higher capacities for ethanol and acetaldehyde clearance, which may contribute to the protective phenotype

The influx of calcium then activates proteases that cleave proglucagon peptides, ultimately boosting GLP-1 production

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