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Epub 2016 Dec 14

Key long-term adaptations include: Improved mitochondrial efficiency, allowing cells to generate energy more reliably Reduced chronic inflammation, which otherwise contributes to fatigue Smoother blood sugar curves, preventing post-meal energy crashes Enhanced fat oxidation, providing more consistent fuel between meals This process is sometimes described as a metabolic reset, and like any reset, it requires an adjustment phase

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Based on the results, ten Kulve and colleagues (2015) argue that endogenous GLP-1 is involved in satiating effects after meal intake in obese patients with T2D (ten Kulve et al

TRIM29 negatively regulates p53 via inhibition of Tip60