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There is no strong evidence of kidney damage in healthy individuals using normal doses

The specific inhibitor of ferroptosis, known as ferrostatin-1 (Fer-1), exerts a suppressive effect on osteoblast susceptibility to ferroptosis by elevating the expression of GPX4, thereby rescuing their osteogenic function and effectively halting the progression of peri-prosthesis osteolysis

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The way Id summarize all of this is that our findings are best thought of as signals worth investigating further, not conclusions