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In conventional one, the pituitary gland triggers hepatic GH cells to liberate GHR into circulation via transcription factors induced by IGF, GH, and GHR

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found that a low-iLe diet can increase liver sensitivity to insulin, increase energy expenditure, and activate the FGF21-UCP1 axis

But during treatment, if the toxins build up in the body faster than the liver can process them, hepatotoxicity may occur [139]