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These morphological improvements suggest a potential mechanism for mitigating vascular dropout and capillary degeneration in glaucomatous conditions, even if direct, short-term neuroprotection was not observed [60]

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(2022) have explored that the -elemene integrated with erlotinib enhanced the cytotoxicity of erlotinib against EGFR mutated primary EGFR-TKI resistant NSCLC cells, and diminished the content of ferroptosis negative regulatory proteins (glutaminase, FTH1, Nrf2, Slc7a11 and GPX4), added ROS and lipid peroxidation levels, and accomplished anti-tumor effects through ferroptosis

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