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By the 1970s, one hormone was identified as an incretin candidate: Gastric Inhibitory Polypeptide (GIP) , discovered in 196970, which was found to stimulate insulin secretion (and renamed Glucose-Dependent Insulinotropic Polypeptide ) ( [30] )

This, in turn, increases peroxisome proliferator-activated receptor-gamma coactivator-1 (PGC1-), stimulating localized energy consumption [27, 47, 104]

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