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Data showed that propionate triggered the release of PYY and GLP-1, with both acute (10 g) and chronic (10 g/day) administration

The rats were randomly divided into 6 experimental treatment groups including: 1) the control group, 2) the diabetes control, 3) the Noopept control, 4) diabetes and Noopept treatment, 5) diabetes and insulin treatment, and 6) diabetes with Noopept and insulin treatment [1]

Profiling metabolites associated with the gut-brain-kidney axis, such as uremic toxins, can offer insights into disease mechanisms and identify biomarkers for early diagnosis and disease monitoring (Yang et al., 2018)

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