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There might be a compensatory relationship between incretin receptors and how GIP-receptor antagonism can enhance GLP-1 receptor activity

According to one survey, a net 30% of people who quit a GLP-1 medication actually eat more calories than they did before they began the course, leading to what has been termed the Ozempic rebound

Ovid Technologies (Wolters Kluwer Health)

And if we dont know if their current protein intake is not being well broken down and absorbed and able to get where its supposed to go, and then we tell someone to significantly increase their protein intake, we may significantly exacerbate those issues

The mechanism appears to be central, BRP acts on neurons in the hypothalamus, the brain region that integrates satiety, hunger, and energy-balance signaling