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One is that Grx1 and Trx2 are involved in repairing metalloenzymes but that alternative routes of reduction are available in well-fed cells
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The arachidonic acid-mediated depletion of PUFA-PLs severely counteracts ferroptosis in a number of cell lines, and a similar phenomenon is observed upon the pharmacological inhibition or genetic inactivation of the acyl-coenzyme A synthetase long chain, which is a requirement for the synthesis of PUFA-PLs
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