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It can also activate extracellular signal-regulated kinase (ERK) and c-Jun N-terminal kinase (JNK), upregulating the proliferation-related gene expression in immune cells

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Vanderlelie and Perkins [7] evaluated the oxidative stress at the end of the reproduction in rats in placental and liver tissues, but they do not describe the alteration of GP activity, resulting from the coordination of tissue GSH reductase and GSSG peroxidase enzyme activities in both selenium deficiency and L-NAME oral administration

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