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Figure 4 shows the forest plots of the six CVOTs that reported the evaluation of MACE risk as a sub-analysis of patients with T2DM divided according to the presence or absence of CV disease at baseline, respectively

The two mutated mouse models represent an unbound form of IGF-1 with impaired post-translational control and increased bioavailability

This better explains that the condition is a whole-body hormonal and metabolic issues

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