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Furthermore, evidence indicates that NRF2 can transcriptionally upregulate CHAC1, as observed in ferroptosis-resistant melanoma cells where CHAC1 induction occurs independently of ER stress (Gagliardi et al., 2019) and modestly at the promoter level ( de novo GSH synthesis (Kre et al., 2023)

However, this phosphorylation was blocked by the GLP-1-R-Antagonist Exendin(9-36), emphasizing the role of GLP-1-mediated signaling [16]