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Interestingly, pretreatment with the antioxidant N -acetyl cysteine (NAC), a precursor of GSH, prevented the lipopolysaccharide induced changes in the biochemical markers of oxidative stress in male fetuses, and the delayed detrimental effects in male offspring, completely restoring both LTP in the hippocampus and spatial recognition performance (Lant et al

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They can no longer produce ATP efficiently, forcing the cells into a toxic, low-energy state

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