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The role of Rac1 in spine morphology was also shown in many other studies by increasing Rac1 activity via knockdown of upstream negative regulators (e.g., Bcr, alpha-chimaerin and p250GAP), expression of constitutively active Rac1, or neurotrophic factor BDNF treatment (Tashiro and Yuste, 2004

Reactions were performed with 5 M histone H3 (histone L-arginine) substrate, 1 M radiolabeled S-adenosyl-L-[methyl- 3 H] methionine (SAM) co-substrate, and recombinant human PRMT3 enzyme

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10.1038/s41598-024-78745-0 156

Reconstitution Required: This kit does not arrive pre-mixed