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ALCAR limits PCa induced pro-angiogenic features and regulates CXCL8-CXCR1/2, CCL2-CCR2 and CXCL12-CXCR4 axis in endothelial cells The vascular endothelial growth factor (VEGF) and CXCL8, a CXC inflammatory chemokine, have been demonstrated to induce angiogenesis and promote progression of PCa [44, 45]

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Patients with multiple comorbidities, especially those with limited treatment options, are significantly more likely to initiate GLP1 therapy

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