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In contrast, injections of PYY3-36 into the third, lateral or fourth cerebral ventricle, the paraventricular nuclei or the hippocampus in rodents potently stimulated food intake and feeding behavior by enhancing expression of NPY/AgRP in neurons of ARC

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GLP-1 receptor activation suppresses IL-1 beta through multiple pathways, including direct NF-kB inhibition and reduced inflammasome activation

Our findings revealed that ITIH4 is cleaved during HAE attacks, depleting intact ITIH4 in plasma