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Mitochondrial targeting in neurodegenerative diseases Mitochondrial abnormalities, such as decreased respiratory capacity, increased mitochondrial fragmentation, and imbalanced mitochondrial fission/fusion, have been identified in the brains of individuals with AD prior to the deposition of pathological -amyloid plaques

The same study showed at 6 months there was a 30% increase in whole blood, 35% increase in red blood cells and a 260% increase in buccal cell glutathione levels

Chronic metformin treatment decreases cardiac injury during ischemia-reperfusion by attenuating endoplasmic reticulum stress with improved mitochondrial function

These findings suggest that selenomethyl selenocysteine may enhance the responsiveness of tumor tissues to anticancer agents by reducing HIF-1 expression in mice bearing tumors, thereby potentially improving the efficacy of cancer treatments (151)

Its levels can be considered a reliable physiological indicator