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Through the activation of Nrf2, SFN inhibited hepatic glucose production and decreased the expression of key enzymes in gluconeogenesis in HFD-induced diabetic rats, and reversed the gene expression profiles of the hepatic disease signature of diabetes

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Die Ergebnisse zeigten, dass GHK-Cu direkt an SIRT1 bindet und einen Protein-Komplex bildet, wobei die interagierenden Aminosaeurereste GLU-230 und ASN-226 identifiziert wurden Li et al., 2025