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We confirmed a strong inverse correlation between beta cell mass and glucose intolerance, consistent with clinical observations that OGTT or continuous glucose monitoring (CGM)-derived metrics can predict symptomatic onset in at-risk individuals once beta cell loss has crossed a critical threshold required for metabolic control [66]

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When this loop is disrupted at any point, either in terms of timing, duration, or magnitude of feedback impulse signaling, hypothalamic loss of feeding is lost, resulting in obesity