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This broad anti-inflammatory mechanism explains the efficacy of GHK-Cu across various inflammation models
The mechanism of toxicity of copper at the cellular level may be a combination of several different copper-sensitive processes that include inhibition of cytosolic enzymes and pro-oxidant effects that damage plasma membrane function and alter oxidative phosphorylation in mitochondria
doi: 10.1159/000485132
J Immunol 202(8):21772187
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