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Whether triggered by DHT sensitivity, sebaceous gland hyperactivity, oxidative stress, environmental aggressors, or autoimmune dysregulation, inflammation around the hair follicle creates a hostile microenvironment in which normal follicular cycling cannot be sustained

Guo XY, Liu XJ, Hao JY

This pattern of accelerating improvement is consistent with the hair growth cycle new follicles need multiple growth cycles to produce visible improvement

Research in animal models shows enhanced collagen deposition and reduced healing time in injury sites treated with thymosin fragments [11]

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