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The von Hippel-Lindau (VHL) protein plays a central role in the oxygen-sensing pathway promoting HIF proteosome-mediated degradation during normoxia

It supports digestive enzymatic processes and helps with nutrient absorption

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It achieves this by enhancing blood flow through angiogenesis, the formation of new blood vessels, which is essential for supplying nutrients and oxygen to injured areas and removing waste and inflammatory byproducts[2][5]