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doi:10.1016/j.tox.2022.153136

That conversion is where mistakes happen

Human Tregs clones produced increased interleukin-10 (IL-10) when treated with thapsigargin, an activator of ER stress and UPR, in an eIF2 phosphorylation-dependent manner ( + T cells differentiated under regulatory conditions in a high oxidative environment ( Whats more, prolonged ER stress and a defective UPR may lead to the activation of inflammatory transcriptional programs and the release of proinflammatory cytokines, generating further ER stress and oxidative stress

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