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The research findings indicated that NAC conferred neuroprotection via two separate compensatory mechanisms: (1) the decrease in -synuclein overexpression, and (2) the modification of NFB translocation from the nucleus to the cytoplasm, thereby preventing the activation of subsequent pro-inflammatory genes

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The cDNA templates were applied to a real-time PCR instrument (Applied Biosystems 7500, United States) for amplification using specific primers for SIRT-1 and CYP2E1

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