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Interestingly, a study reported that a rapid increase in proBDNF following status epilepticus induced by pilocarpine may be in part due to decreasing its cleavage due to a decrease in tissue plasminogen activator and an increase in plasminogen activator inhibitor-1 (PAI-1), an inhibitor of extracellular and intracellular cleavage, and that proBDNF is initially enhanced in response to the acute phase of epileptogenesis (Thomas et al

Quercetin attenuates oxidative damage by stimulating Nrf2 translocation to the nucleus and concurrently inhibiting NF-Bmediated inflammatory signaling, which together mitigate ROS-induced apoptosis (86, 87)

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