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Similarly, pharmacological inhibition of HSL and knockdown of PNPLA2 reduced the expression of known PPAR target genes in which LDs were shown to be a source of PPAR and PPAR ligands that could be generated within minutes of stimulating lipolysis (3)

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It stimulates angiogenesis to improve blood supply to damaged tissues, enhances fibroblast activity for superior collagen formation, and modulates inflammatory cascades to optimize the healing environment