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PARIS induced defects in mitochondrial biogenesis drive dopamine neuron loss under conditions of parkin or PINK1 deficiency

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Although both antioxidants used as pre-treatment (Mel and GSH) mitigated the harmful effects of CARB, Mel appeared to exert comparatively stronger inhibitory effects on apoptotic (Cas-3) and inflammatory (NF-B, TNF-, IL-6) responses

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