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1d was driven by bioisosteric replacement of the fragments bound to subpockets A-C, scaffold-hopping, de novo design, and medicinal chemistry and was facilitated by SZMAP/GamePlan 37 , vBrood 38 , and MOE software 39

But on the other hand, for cysteine-deprived cells, the catabolism of Gln promoted the synthesis of PUFA, and its decomposition products such as -ketoglutaric acid could also increase the accumulation of lipid peroxides

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