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This mechanism may explain the heightened vulnerability to fractures observed in hEDS patients
Renguet E, Ginion A, Gelinas R, Bultot L, Auquier J, Robillard Frayne I, Daneault C, Vanoverschelde JL, Des Rosiers C, Hue L, Horman S, Beauloye C, Bertrand L (2017) Metabolism and acetylation contribute to leucine-mediated inhibition of cardiac glucose uptake
(e.g., Redesign Health, PE firms doing roll-ups) Operators make GLP-1 care scalable and profitable, bridging the gap between science and patient experience
observed additive effects likely reflect convergence on different limiting factors of tissue repair rather than pathway synergy