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568 constructed a biologically inspired PROTAC against TRIM24 by coating the poly lactic-co-glycolic acid (PLGA) nanoparticles containing PROTAC degrader with M2 macrophage membrane (MELT) for atherosclerosis

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Combining network pharmacology and molecular docking, Akt was identified as the key therapeutic target of LC in cardiomyopathy and demonstrated good binding affinity with LC