The absence of BRAF, NRAS, TERT mutations, loss of PRKAR1A expression, the presence of psammoma bodies, and paravertebral location allow differentiation of MMPN from melanoma metastases [50]
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PubMed DOI Sikiric P, Rucman R, Turkovic B, et al
Keywords: oxidative damage, Parkinsons disease, Huntingtons disease, 3-nitropropionic acid, MPTP, R6/2 HD mice Introduction There is substantial evidence that mitochondrial dysfunction and bioenergetic abnormalities play a role in the pathogenesis of neurodegenerative disease (Lin and Beal 2006)
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