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Notably, neurexin-3-SS4+ selective splicing propensity was induced in the hippocampus after transient CVS, and the mechanism of the subsequent impact of this effect may be related to the restriction of fast receptor trafficking induced by postsynaptic AMPA receptor anchoring, which is closely associated with reduced synaptic plasticity [76]

It acts as an obligate cofactor for the carnitine palmitoyltransferase (CPT) enzyme system, which shuttles long-chain fatty acids across the inner mitochondrial membrane for -oxidation [1]

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